Plastic Surgery – Surgical Case Studies

May 13, 2025

Case Study: Neurofibromatosis and Chronic Leg Ulcers


I. Concept Digestion

1. Differentiation of Ulcer Types

Feature Venous Ulcer Arterial Ulcer Lymphatic Ulcer Malignant Ulcer
Location Medial malleolus (gaiter area) Toes, heels, pressure points Dorsum of foot Anywhere, esp. chronic ulcer sites
Appearance Shallow, irregular, granulating Punched out, deep, necrotic Shallow, oozing Raised, everted edges, necrotic base
Pain Mild, relieved by elevation Severe, worse on elevation Mild to moderate Painless or neuropathic pain
Surrounding Skin Hemosiderin deposits, eczema Cold, hairless, shiny Thickened, hyperkeratotic Indurated, pigmented, satellite nodules
Pulses Present Absent/Reduced Normal May be normal or reduced
Discharge Serous or purulent Minimal Proteinaceous lymph Bloody, foul-smelling
Other Signs Varicose veins Claudication, rest pain, gangrene Lymphedema, peau d’orange Regional nodes, systemic signs

III. Clinical/Applied Simulation – Consultant Round

Consultant: "You’re reviewing a leg ulcer—defaulting to 'venous' without thinking? That’s amateur. Walk me through your reasoning. I’ll fail fluff."

You (student):

  • Pain: Is it worse on walking (arterial) or relieved by elevation (venous)?
  • Location: Medial ankle (venous) vs toes/heel (arterial)?
  • Edges: Irregular = venous, punched out = arterial, everted = malignant.
  • Discharge: Serous vs lymph-rich vs bloody/foul?
  • Pulses: Check all – absent is arterial clue.
  • Systemic features: Weight loss, fever? Think malignancy.

Consultant: "You missed ulcer age, prior trauma, neuropathy, and lymphadenopathy. You want to be a surgeon? Redo it in SOAP."


IV. Neurofibromatosis

Definition:

  • NF1 (von Recklinghausen) – Chromosome 17 – Neurofibromin mutation.
  • NF2 – Chromosome 22 – Merlin mutation.

Clinical Features of NF1:

System Manifestation
Skin Café-au-lait spots (≥6), axillary freckling
Neuro Seizures, learning disabilities
Eye Lisch nodules (iris hamartomas)
Tumors Neurofibromas (cutaneous/plexiform), gliomas
MSK Scoliosis, pseudoarthrosis, bone dysplasia
Endocrine Pheochromocytoma, HTN

V. Clerking Template – Neurofibromatosis + Leg Ulcer

Identification:

  • Name, Age, Sex, Occupation
  • Presenting complaint

HPI:

  • Duration of bumps/ulcers
  • Pain, discharge, prior similar issues
  • Neurological Sx: vision, seizures, learning
  • Ulcer: type, progression, treatment history

PMH:

  • Seizures, tumors, HTN, previous surgeries

Family History:

  • Neurofibromatosis features in relatives

Social:

  • Stigma, occupation, education

ROS:

  • CNS: headache, weakness, seizures
  • CVS: palpitations, BP history
  • MSK: limb deformities, scoliosis

Exam:

  • Skin: Café-au-lait, freckling, neurofibromas
  • Eyes: Lisch nodules
  • Neuro: Focal deficits
  • MSK: Pseudoarthrosis, scoliosis
  • Ulcer: Type, base, edges, discharge, pulses

VI. Monitoring Skin Flap Viability

Parameter Interpretation
Color Pink = good, Pale = ischemia, Blue = congestion
Cap Refill Normal <2s, delayed = ischemia
Temperature Cool = ischemic, compare with adjacent skin
Bleeding Edge Presence = good perfusion
Area of Discoloration Progression = poor perfusion

VII. Why Not Graft Over Medial Malleolus?

  • Grafts require granulation tissue – bone/tendon exposure = fail
  • Flaps bring their own blood supply
  • Better durability, contouring over pressure zones
  • Higher success rates in ischemic or infected beds

VIII. Why Use Hb, Not PCV, for Surgical Planning?

Reason Explanation
Hb = O₂ carrying power Direct measure of oxygen transport
PCV misleads Affected by hydration status
Acute blood loss PCV lags behind, Hb reflects real deficit
Volume status shift PCV rises with dehydration falsely

Consultant: "Stop quoting PCV like it’s gospel. Hb tells you if your patient will bleed out or breathe post-op. PCV is for clerks, not surgeons."


IX. Pre-Operative Investigations

Test Purpose
FBC Anemia, infection, platelet status
U&E Renal function, electrolytes
PT/INR/aPTT Bleeding risk
HbA1c/RBS Glycemic control
ECG Baseline rhythm, ischemic changes
CXR Pulmonary pathology, cardiomegaly
Crossmatch Prepare for transfusion
HIV/HBV/HCV Status, theatre risk, consent implications
Chest function tests Indicated in COPD/asthma patients
Imaging For ulcer depth, osteomyelitis, vascular supply

X. From History – Ruling Out Types of Ulcers

Key Differentiating Questions

Ulcer Type Focused Questions
Venous - Do your legs swell at the end of the day?
- Does elevating the leg relieve pain?
- History of varicose veins?
- Any brownish skin discoloration?
Arterial - Do you have leg pain when walking (claudication)?
- Is the pain worse at night or when legs are elevated?
- Do your feet feel cold or numb?
- Do you smoke or have diabetes/hypertension?
Lymphatic - Do you have a long-standing swelling of the limb?
- Any history of recurrent cellulitis?
- Milky or clear discharge from the ulcer?
- Travel to filariasis-endemic areas?
Malignant - Has the ulcer been present for many years?
- Is the base of the ulcer bleeding or foul-smelling?
- Has it changed in size or appearance recently?
- Any nearby hard lumps (lymph nodes)?

XI. Simulation – The Angry Consultant

Consultant:
"Why the hell would you say 'venous' without asking about claudication? If that patient has rest pain and you slap a compression bandage on them, you’re writing the amputation request. Again—what. specific. history. question. rules out malignancy?"

Expected Student Response:
"Chronicity, foul discharge, and progressive change in size or bleeding are suspicious. Ask about lymphadenopathy, prior trauma, and systemic signs like weight loss."

Consultant:
"Better. But if you don’t examine pulses and don’t biopsy a suspicious ulcer—you deserve your malpractice suit. Next!"


XIII. Clinical Monitoring – Flap Post-Op Care

Mnemonic: "The FLAP CODE"

Code Letter Meaning Explanation
F Flush of Color Pink = good, Pale = ischemia, Dusky = venous congestion
L Local Temp Compare with surrounding skin – cold = compromise
A Area of Discoloration Progressive darkening = early necrosis
P Peripheral Bleeding Prick the edge – absence of bleeding = bad perfusion
C Capillary Refill Normal <2s = viable
O Odor/Discharge Foul smell = infection = decompensation
D Doppler Signal Confirm arterial inflow
E Edge Tension Excess pressure = poor flow; flap too tight?

Consultant Warning:
“You lose a flap, you fail the rotation. I want round-the-clock charting. No guesswork.”


XIV. Integration Summary (For Visual Notes / Flashcards)

Markdown Table: Ulcer Differentiation & Management

Ulcer Type Key Feature Diagnostic Test Key History Question Treatment Approach
Venous Gaiter zone, edema Duplex US Worse with standing? Varicosities? Compression, elevation, skin care
Arterial Toes, punched out ABI, Doppler Claudication? Worse at night? Revascularization, avoid compression
Lymphatic Dorsum, lymphedema Lymphoscintigraphy Chronic swelling? Travel history? Compression, manual drainage, hygiene
Malignant Everted edge, bleeding Biopsy, Imaging Chronic ulcer with changes? Weight loss? Wide excision, oncology input

OSCE-Style Flash Drills – Ulcer, NF, Flap Viability

STATION 1: Ulcer Diagnosis

Scenario:
A 60-year-old man presents with a 6-month history of a non-healing ulcer over the medial malleolus.

Q1: What is the most likely cause?
A: Venous ulcer.

Q2: What history supports this?
A: Chronicity, location (gaiter area), worse with standing, relief with leg elevation, hx of varicose veins.

Q3: What examination signs do you look for?
A: Hemosiderin staining, lipodermatosclerosis, varicose veins, edema, warm skin, granulating base.

Consultant Critique:
“If you didn’t check pulses before applying compression—you've just necrosed the leg. Always rule out arterial disease!”


STATION 2: Rule Out Arterial Ulcer

Scenario:
Same patient. You suspect arterial involvement.

Q1: What symptoms point to arterial ulcers?
A: Intermittent claudication, rest pain, cool skin, reduced/absent pulses, ulcers on pressure points like toes.

Q2: What test confirms your suspicion?
A: Ankle-Brachial Index (ABI). <0.9 suggests arterial insufficiency.

Consultant Critique:
"ABI first. Doppler next. If you don't check this before compression, you're committing iatrogenic murder."


STATION 3: Neurofibromatosis History Taking

Scenario:
A 25-year-old male presents with multiple skin swellings and pigmentation.

Q1: What system-based history must you ask?
A:

  • Neuro: seizures, headaches, learning disability
  • Derm: onset and change in lesions, pruritus
  • Ortho: bone pain, scoliosis
  • Eyes: vision issues, Lisch nodules
  • FHx: family members with similar signs

Q2: What’s your provisional diagnosis?
A: Neurofibromatosis Type 1 (NF1)

Q3: What features meet NIH criteria?
A: ≥2 of:

  • ≥6 café-au-lait spots
  • Axillary/inguinal freckling
  • ≥2 neurofibromas or 1 plexiform
  • Optic glioma
  • ≥2 Lisch nodules
  • Osseous lesions (e.g., sphenoid dysplasia)
  • First-degree relative with NF1

Consultant Critique:
"If you didn't screen for learning disabilities or optic issues, you've missed 40% of the complications. Unacceptable."


STATION 4: Skin Flap Monitoring Post-Op

Scenario:
Post-op day 1 following flap coverage of a medial malleolus wound.

Q1: What 5 things must be monitored hourly?
A:

  • Color
  • Temperature
  • Capillary refill
  • Edge bleeding
  • Doppler signal

Q2: What’s an early sign of venous congestion?
A: Purple/dusky color with brisk bleeding

Q3: How would you test arterial flow?
A: Doppler signal and capillary refill <2s.

Consultant Critique:
"Monitoring is your life insurance. If you miss duskiness, necrosis is next. Flaps don’t forgive mistakes."


STATION 5: Graft vs Flap

Scenario:
You're deciding between a skin graft and a flap over the medial malleolus.

Q1: Why not a graft here?
A: Poor vascularity on bony prominence, lack of granulation tissue, exposed tendon/bone.

Q2: What advantages does a flap offer?
A: Brings its own blood supply, better for coverage of poorly vascularized tissue, more durable.

Mnemonic Recap:
Battle at Malleolus Fortress – Grafts need fertile fields; Flaps bring blood and army.

Consultant Critique:
"If you slap a graft on bone, it’s like planting seeds on rock—don’t waste the theatre time."


STATION 6: PCV vs Hb – Surgical Fitness

Scenario:
Pre-op anemia screen for a patient with a chronic ulcer needing skin coverage.

Q1: Why is Hb better than PCV?
A:

  • Hb measures oxygen-carrying capacity directly
  • PCV is volume-based, affected by hydration status

Q2: What’s a misleading situation where PCV fails?
A: Dehydration (false high PCV, normal Hb) or fluid overload (false low PCV).

Consultant Critique:
"Rely on PCV alone and you might cut into a hypoxic patient. Use Hb—end of discussion."


STATION 7: Pre-op Investigations – Interpretation

Scenario:
You’re screening a patient for elective flap surgery.

Investigations & Purpose:

Investigation Purpose
FBC Anemia, leukocytosis (infection), platelet count
U&E Electrolyte balance, renal function (anesthesia safety)
RBS/FBS Diabetic control, wound healing potential
ECG Cardiac fitness for anesthesia
Coagulation Panel Bleeding risk (esp. important for flap survival)
HIV/Hep B/C Infection risk, immunosuppression
Chest X-Ray Pulmonary evaluation if age/comorbidity suggestive

Consultant Critique:
"Surgery is controlled trauma. If you don’t evaluate the battlefield—don’t enter it."


Anki Flashcards – OSCE: Ulcers, Neurofibromatosis, Flaps


Q: What is the most likely cause of a chronic ulcer over the medial malleolus?
A: Venous ulcer.


Q: What history supports a diagnosis of venous ulcer?
A: Chronicity, gaiter area location, worsens on standing, improves with elevation, hx of varicose veins.


Q: What physical signs support venous ulcers?
A: Hemosiderin staining, lipodermatosclerosis, edema, varicosities, warm granulating ulcer base.


Q: What must you rule out before applying compression therapy in venous ulcer?
A: Arterial insufficiency (via pulse exam and ABI).


Q: What symptoms point to an arterial ulcer?
A: Claudication, rest pain, cool skin, diminished/absent pulses, ulcer on toes or pressure points.


Q: What is the most useful bedside test for arterial disease?
A: Ankle-Brachial Index (ABI); <0.9 indicates arterial insufficiency.


Q: What 7 system-based questions must you ask when evaluating a patient for Neurofibromatosis?
A:

  1. Seizures
  2. Headaches
  3. Vision changes
  4. Learning difficulty
  5. Skin lesion history
  6. Bone pain/deformity
  7. Family history of similar lesions

Q: What are the NIH diagnostic criteria for NF1 (name 4 out of 7)?
A:

  • ≥6 café-au-lait spots
  • Axillary/inguinal freckling
  • ≥2 neurofibromas or 1 plexiform
  • Optic glioma
  • ≥2 Lisch nodules
  • Osseous lesions
  • 1st-degree relative with NF1

Q: What five parameters should be monitored post-operatively in a skin flap?
A:

  1. Color
  2. Capillary refill
  3. Temperature
  4. Edge bleeding
  5. Doppler signal

Q: What early sign indicates venous congestion in a flap?
A: Dusky/purple color with brisk bleeding on needle prick.


Q: Why is a flap preferred over a graft in wounds over the medial malleolus?
A: Grafts fail over bony prominences or tendon without paratenon. Flaps bring their own vascular supply.


Q: When is a skin graft contraindicated?
A:

  • Exposed bone/tendon
  • Poor vascularity
  • No granulation tissue

Q: Why is Hb concentration better than PCV in surgical planning?
A: Hb directly reflects oxygen-carrying capacity; PCV is volume-dependent and can be misleading.


Q: In what situations is PCV unreliable?
A:

  • Dehydration (falsely high PCV)
  • Overhydration (falsely low PCV)

Q: List 7 essential pre-operative investigations and their purposes.
A:

  • FBC: anemia, infection, platelets
  • U&E: electrolyte balance, kidney function
  • RBS: diabetes control
  • ECG: cardiac fitness
  • Coagulation screen: bleeding risk
  • HIV/HepB/C: infection risk
  • CXR: pulmonary baseline if indicated