PRECIOUS IKHIANOSE AITANUN · CLINICAL JOURNAL Daily Entry Trauma Care – Shock Overview May 14, 2025 Daily Serious Detailed mindmap on shock CLASS DIAGRAM presents withtreated byShock+Acute circulatory failure+Cellular dysfunction+Life-threatening+evaluatePatient()+restoreTissuePerfusion()HypovolaemicShock+Collapse of cardiac preload+Blood / Plasma / ECF loss+controlBloodLoss()+replaceFluid()CardiogenicShock+Pump failure+MI / Arrhythmias / Cardiomyopathy+improveContractility()DistributiveShock+Collapse of peripheral resistance+Vasodilation / pooling+restoreVascularTone()ObstructiveShock+Restricted blood outflow+PE / Tamponade / Pneumothorax+removeObstruction()EndocrineShock+Mixed presentation+Hormonal imbalance+correctHormones()SepticShock+Bacterial endotoxins+Cytokine activation+administerAntibiotics()AnaphylacticShock+Type 1 hypersensitivity+Histamine release+administerAdrenaline()NeurogenicShock+Unopposed vagal tone+Spinal cord injury+administerVasopressors()ClinicalFeatures+Cold skin / Rapid pulse+Type-dependent signs+Oliguria / CNS disturbanceManagement+O2 / IV access / Monitor+Type-specific therapy+Prevent DIC / MODS FLOW CHART SHOCKAcute circulatory failure compromising tissue perfusionIntroductionTypes of ShockClinical FeaturesINVESTIGATIONSManagementDefinition: Acute circulatory failurecompromising tissue perfusionManifestation of cellulardysfunction due to inadequate perfusionLife-threatening: progresses toirreversible damage if untreatedHypovolaemic ShockCardiogenic ShockDistributive ShockObstructive ShockEndocrine ShockDefinition: Collapse of cardiacpreload due to reduced volumeCausesPathophysiologyBlood LossInternal hemorrhageExternal hemorrhageLoss of PlasmaExtensive burnsPeritonitisLoss of Extracellular FluidIntestinal obstructionDiarrhea and vomitingMassive fluid loss intointerstitial tissuesSepsisAcute pancreatitisEarly StageReduced circulating volumeBaroreceptor responseAdrenal-sympathetic stimulationAdrenaline/Noradrenaline releaseIncreased heart rate and vasoconstrictionMiddle StageContinued sympathetic overactivityActivation of RAASSplanchnic vasoconstrictionBacterial translocationAnaerobic respiration and lactic acidosisLate StageCompensatory mechanisms failCapillary endothelium damageDecreased cerebral blood flowProgressive cerebral dysfunctionDefinition: Pump failure;ventricles fail to pump sufficientlyCausesAcute Myocardial InfarctionArrhythmiasCardiomyopathiesHeart valve problemsMechanical factorsDefinition: Collapse ofperipheral resistanceMechanismsSubtypesVasodilation of arterioles & venulesPeripheral poolingSeptic ShockBacterial endotoxinsCytokine activationEndothelial damageAnaphylactic ShockType 1 hypersensitivity reactionHistamine/vasoactive amine releaseNeurogenic ShockUnopposed vagal toneCervical spinal cord injuryDefinition: Physical restrictionto blood outflowCausesPulmonary embolismCardiac tamponadeTension pneumothoraxCombination of hypovolaemic,cardiogenic, and distributive shockCauses: Hormonal imbalancesGeneral FeaturesCold, white, clammy patientRapid thready pulseIncreased respiratory rateFall in arterial BPReduced urinary outputSeptic Shock contrastHot, flushed skinBounding pulseSevere SignsWeak, thready pulseCold, clammy skinCollapsed peripheral veinsRapid respirationCNS disturbancesOliguria or anuriaBaseline hemoglobin/hematocritBlood grouping and cross-matchingSerum electrolytes, urea, creatinineArterial blood gasesSerum lactateBlood glucoseBlood culturesAim: Restore tissue perfusion and oxygenationGeneral StepsSpecific TreatmentComplicationsOxygen therapyVenous access and fluid resuscitationContinuous assessmentTreat primary problemHypovolaemic Shock TreatmentIdentify cause of fluid lossControl blood lossFluid replacementCardiogenic Shock TreatmentCautious fluid resuscitationImprove myocardial contractilityTreat underlying pathologyAnaphylactic Shock TreatmentAdrenalineHydrocortisone & ChlorpheniramineIV fluidsNeurogenic Shock TreatmentSecure airwayFluid resuscitationVasoconstrictors if neededSeptic Shock TreatmentAdequate fluid resuscitationAppropriate antibioticsSurgical elimination of infection sourceDisseminated Intravascular CoagulationMultiple Organ Dysfunction Syndrome ← Previous Plastic Surgery – Surgical Case Studies ← All journal entries